Protein kinase C-dependent diacylglycerol formation is mediated via Ca2+ / calmodulin in parotid cells
- 1 July 1991
- journal article
- Published by Elsevier in European Journal of Pharmacology: Molecular Pharmacology
- Vol. 207 (3) , 175-181
- https://doi.org/10.1016/0922-4106(91)90028-g
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Hormonal stimulation of diacylglycerol formation in hepatocytes.Published by Elsevier ,1989
- Activation of protein kinase C alters the interaction of α2‐adrenoceptors and the inhibitory GTP‐binding protein (Gi) in human plateletsFEBS Letters, 1989
- Mechanisms whereby insulin increases diacylglycerol in BC3H-1 myocytesBiochemical Journal, 1988
- Properties of receptor-controlled inositol trisphosphate formation in parotid acinar cellsBiochemical Journal, 1985
- Inositol trisphosphate, a novel second messenger in cellular signal transductionNature, 1984
- Isoquinolinesulfonamides, novel and potent inhibitors of cyclic nucleotide-dependent protein kinase and protein kinase CBiochemistry, 1984
- Inositol trisphosphate and diacylglycerol as second messengersBiochemical Journal, 1984
- Renewed interest in the polyphosphoinositidesTrends in Biochemical Sciences, 1984
- Evidence for de novo synthesis of phosphatidylinositol coupled with histamine release in activated rat mast cellsBiochemical and Biophysical Research Communications, 1983
- [17] Naphthalenesulfonamides as calmodulin antagonistsPublished by Elsevier ,1983