Altered SDF-1/CXCR4 axis in patients with primary myelofibrosis and in the Gata1low mouse model of the disease
Open Access
- 29 February 2008
- journal article
- Published by Elsevier in Experimental Hematology
- Vol. 36 (2) , 158-171
- https://doi.org/10.1016/j.exphem.2007.10.001
Abstract
No abstract availableKeywords
This publication has 98 references indexed in Scilit:
- Pivotal contributions of megakaryocytes to the biology of idiopathic myelofibrosisBlood, 2007
- Hematopoietic Progenitor Cells (HPC) from Mobilized Peripheral Blood Display Enhanced Migration and Marrow Homing Compared to Steady-State Bone Marrow HPCExperimental Hematology, 2007
- A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia veraNature, 2005
- CLIP Identifies Nova-Regulated RNA Networks in the BrainScience, 2003
- Nova Regulates GABAA Receptor γ2 Alternative Splicing via a Distal Downstream UCAU-Rich Intronic Splicing EnhancerMolecular and Cellular Biology, 2003
- Listening to silence and understanding nonsense: exonic mutations that affect splicingNature Reviews Genetics, 2002
- The tetranucleotide UCAY directs the specific recognition of RNA by the Nova K-homology 3 domainProceedings of the National Academy of Sciences, 2000
- Sequence-Specific RNA Binding by a Nova KH DomainCell, 2000
- Function of the chemokine receptor CXCR4 in haematopoiesis and in cerebellar developmentNature, 1998
- Nova, the paraneoplastic Ri antigen, is homologous to an RNA-binding protein and is specifically expressed in the developing motor systemNeuron, 1993