p38 Kinase-mediated Transactivation of the Epidermal Growth Factor Receptor Is Required for Dedifferentiation of Renal Epithelial Cells after Oxidant Injury
Open Access
- 1 June 2005
- journal article
- Published by Elsevier
- Vol. 280 (22) , 21036-21042
- https://doi.org/10.1074/jbc.m413300200
Abstract
No abstract availableKeywords
This publication has 56 references indexed in Scilit:
- Requirement of the epidermal growth factor receptor in renal epithelial cell proliferation and migrationAmerican Journal of Physiology-Renal Physiology, 2004
- Oxidative and Osmotic Stress Signaling in Tumor Cells Is Mediated by ADAM Proteases and Heparin-Binding Epidermal Growth FactorMolecular and Cellular Biology, 2004
- Metalloprotease-dependent ErbB ligand shedding in mediating EGFR transactivation and vascular remodellingBiochemical Society Transactions, 2003
- Multiple G-protein-coupled receptor signals converge on the epidermal growth factor receptor to promote migration and invasionOncogene, 2003
- Src Family Protein-tyrosine Kinases Alter the Function of PTEN to Regulate Phosphatidylinositol 3-Kinase/AKT CascadesJournal of Biological Chemistry, 2003
- p38 Mitogen-Activated Protein Kinases on the Body Surface – A Function for p38δJournal of Investigative Dermatology, 2003
- Stress kinase p38 mediates EGFR transactivation by hyperosmolar concentrations of sorbitolJournal of Cellular Physiology, 2002
- ATP Depletion Of Tubular Cells Causes Dissociation of the Zonula Adherens and Nuclear Translocation of β-Catenin and LEF-1Journal of the American Society of Nephrology, 2002
- Regeneration of Renal Proximal Tubule Cells in Primary Culture Following Toxicant Injury: Response to Growth FactorsToxicology and Applied Pharmacology, 1995
- Expression of transforming growth factor-α and epidermal growth factor receptor in human fetal kidneysMolecular and Cellular Endocrinology, 1991