The Effect of Hemorrhagic Hypotension and Retransfusion and 7‐Nitro‐Indazole on rCBF, NOS Catalytic Activity, and Cortical NO Content in the Cata
- 1 November 1994
- journal article
- Published by Wiley in Annals of the New York Academy of Sciences
- Vol. 738 (1) , 348-368
- https://doi.org/10.1111/j.1749-6632.1994.tb21822.x
Abstract
No abstract availableThis publication has 68 references indexed in Scilit:
- Rabbit brain contains an endogenous inhibitor of endothelium-dependent relaxationBritish Journal of Pharmacology, 1990
- Nitric oxide and cyclic GMP formation upon electrical field stimulation cause relaxation of corpus cavernosum smooth muscleBiochemical and Biophysical Research Communications, 1990
- Pressor effect of NG-nitro-L-arginine in pentobarbital-anesthetized ratsLife Sciences, 1990
- Enzymatic formation of nitrogen oxides from L-arginine in bovine brain cytosolBiochemical and Biophysical Research Communications, 1989
- Formation of nitric oxide from L-arginine in the central nervous system: a transduction mechanism for stimulation of the soluble guanylate cyclase.Proceedings of the National Academy of Sciences, 1989
- A specific inhibitor of nitric oxide formation from l‐arginine attenuates endothelium‐dependent relaxationBritish Journal of Pharmacology, 1989
- Endothelium-derived relaxing factor release on activation of NMDA receptors suggests role as intercellular messenger in the brainNature, 1988
- Vascular endothelial cells synthesize nitric oxide from L-arginineNature, 1988
- Nitric oxide release accounts for the biological activity of endothelium-derived relaxing factorNature, 1987
- The obligatory role of endothelial cells in the relaxation of arterial smooth muscle by acetylcholineNature, 1980