Expression of the Prader-Willi gene Necdin during mouse nervous system development correlates with neuronal differentiation and p75NTR expression
- 12 August 2003
- journal article
- research article
- Published by Elsevier in Gene Expression Patterns
- Vol. 3 (6) , 761-765
- https://doi.org/10.1016/s1567-133x(03)00138-8
Abstract
No abstract availableKeywords
This publication has 11 references indexed in Scilit:
- The p75 Neurotrophin Receptor Interacts with Multiple MAGE ProteinsPublished by Elsevier ,2002
- Expression analysis of a novel p75NTR signaling protein, which regulates cell cycle progression and apoptosisMechanisms of Development, 2002
- A Novel Role for p75NTR in Subplate Growth Cone Complexity and Visual Thalamocortical InnervationJournal of Neuroscience, 2002
- Pax6 Regulates the Identity of Embryonic Diencephalic NeuronsMolecular and Cellular Neuroscience, 2001
- Disruption of the mouse Necdin gene results in hypothalamic and behavioral alterations reminiscent of the human Prader-Willi syndromeHuman Molecular Genetics, 2000
- Cellular and Subcellular Localization of Necdin in Fetal and Adult Mouse BrainDevelopmental Neuroscience, 2000
- NRAGE, A Novel MAGE Protein, Interacts with the p75 Neurotrophin Receptor and Facilitates Nerve Growth Factor–Dependent ApoptosisNeuron, 2000
- Enlarged cholinergic forebrain neurons and improved spatial learning in p75 knockout miceEuropean Journal of Neuroscience, 2000
- Neurotrophins and the neuroendocrine brain: different neurotrophins sustain anatomically and functionally segregated subsets of hypothalamic dopaminergic neuronsJournal of Neuroscience, 1995
- Expression of necdin, an embryonal carcinoma-derived nuclear protein, in developing mouse brainDevelopmental Brain Research, 1992