Interferons limit inflammatory responses by induction of tristetraprolin
Open Access
- 15 June 2006
- journal article
- Published by American Society of Hematology in Blood
- Vol. 107 (12) , 4790-4797
- https://doi.org/10.1182/blood-2005-07-3058
Abstract
Interferons (IFNs) are cytokines with pronounced proinflammatory properties. Here we provide evidence that IFNs also play a key role in decline of inKeywords
This publication has 53 references indexed in Scilit:
- Minute numbers of contaminant CD8+ T cells or CD11b+CD11c+ NK cells are the source of IFN-γ in IL-12/IL-18-stimulated mouse macrophage populationsBlood, 2005
- Toll-like receptor control of the adaptive immune responsesNature Immunology, 2004
- Post-transcriptional regulation of proinflammatory proteinsJournal of Leukocyte Biology, 2004
- MAPKAP Kinase 2 Phosphorylates Tristetraprolin on in Vivo Sites Including Ser178, a Site Required for 14-3-3 BindingJournal of Biological Chemistry, 2004
- p38α Mitogen-activated Protein Kinase Sensitizes Cells to Apoptosis Induced by Different StimuliMolecular Biology of the Cell, 2004
- Interferon-γ: an overview of signals, mechanisms and functionsJournal of Leukocyte Biology, 2003
- Transcriptional Regulation of Tristetraprolin by Transforming Growth Factor-β in Human T CellsJournal of Biological Chemistry, 2003
- Impaired On/Off Regulation of TNF Biosynthesis in Mice Lacking TNF AU-Rich ElementsImmunity, 1999
- Targeted Disruption of the Mouse Stat1 Gene Results in Compromised Innate Immunity to Viral DiseaseCell, 1996
- Targeted Disruption of the Stat1 Gene in Mice Reveals Unexpected Physiologic Specificity in the JAK–STAT Signaling PathwayCell, 1996