Fibroblast Growth Factor-21 Regulates PPARγ Activity and the Antidiabetic Actions of Thiazolidinediones
Top Cited Papers
- 1 February 2012
- Vol. 148 (3) , 556-567
- https://doi.org/10.1016/j.cell.2011.11.062
Abstract
No abstract availableKeywords
This publication has 61 references indexed in Scilit:
- Anti-diabetic drugs inhibit obesity-linked phosphorylation of PPARγ by Cdk5Nature, 2010
- Hepatic FGF21 Expression Is Induced at Birth via PPARα in Response to Milk Intake and Contributes to Thermogenic Activation of Neonatal Brown FatPublished by Elsevier ,2010
- FGF21 induces PGC-1α and regulates carbohydrate and fatty acid metabolism during the adaptive starvation responseProceedings of the National Academy of Sciences, 2009
- PPARγ and C/EBP factors orchestrate adipocyte biology via adjacent binding on a genome-wide scaleGenes & Development, 2008
- Inhibition of Growth Hormone Signaling by the Fasting-Induced Hormone FGF21Published by Elsevier ,2008
- Tissue-specific Expression of βKlotho and Fibroblast Growth Factor (FGF) Receptor Isoforms Determines Metabolic Activity of FGF19 and FGF21Journal of Biological Chemistry, 2007
- Endocrine Regulation of the Fasting Response by PPARα-Mediated Induction of Fibroblast Growth Factor 21Published by Elsevier ,2007
- βKlotho is required for metabolic activity of fibroblast growth factor 21Proceedings of the National Academy of Sciences, 2007
- A SUMOylation-dependent pathway mediates transrepression of inflammatory response genes by PPAR-γNature, 2005
- Stimulation of adipogenesis in fibroblasts by PPARγ2, a lipid-activated transcription factorCell, 1994