Depletion of brain histamine produces regionally selective protection against thiamine deficiency-induced lesions in the rat.
- 1 January 2002
- journal article
- Published by Springer Nature in Metabolic Brain Disease
- Vol. 17 (3) , 199-210
- https://doi.org/10.1023/a:1019930206196
Abstract
No abstract availableKeywords
This publication has 39 references indexed in Scilit:
- Evaluation of the Role of NMDA-Mediated Excitotoxicity in the Selective Neuronal Loss in Experimental Wernicke EncephalopathyExperimental Neurology, 1998
- Blood-Brain Barrier Abnormalities in Vulnerable Brain Regions during Thiamine DeficiencyExperimental Neurology, 1995
- Evidence for early blood-brain barrier breakdown in experimental thiamine deficiency in the mouseMetabolic Brain Disease, 1995
- Mast cells in neuroimmune function: Neurotoxicological and neuropharmacological perspectivesNeurochemical Research, 1995
- Histamine‐mediated neuronal death in a rat model of Wernicke's encephalopathyJournal of Neuroscience Research, 1994
- Extracellular Glutamate Is Increased in Thalamus During Thiamine Deficiency‐Induced Lesions and Is Blocked by MK‐801Journal of Neurochemistry, 1993
- Wernickeʼs Encephalopathy Manifested as Korsakoffʼs Syndrome in a Patient With Promyelocytic LeukemiaSouthern Medical Journal, 1990
- Regional changes in brain histamine levels following dietary-induced thiamine deficiency in rats.The Japanese Journal of Pharmacology, 1988
- Effect of Thiamine Deficiency and its Reversal on Cerebral Blood Flow in the Rat. Observations on the Phenomena of Hyperperfusion, “No Reflow,” and Delayed HypoperfusionJournal of Cerebral Blood Flow & Metabolism, 1986
- A new radioenzymatic assay for histamine using purified histamine N-methyltransferaseLife Sciences, 1983