Selective glutathione depletion of mitochondria by ethanol sensitizes hepatocytes to tumor necrosis factor
- 1 December 1998
- journal article
- Published by Elsevier in Gastroenterology
- Vol. 115 (6) , 1541-1551
- https://doi.org/10.1016/s0016-5085(98)70034-4
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- NFkappaB prevents apoptosis and liver dysfunction during liver regeneration.Journal of Clinical Investigation, 1998
- Evidence That the Rat Hepatic Mitochondrial Carrier Is Distinct from the Sinusoidal and Canalicular Transporters for Reduced GlutathioneJournal of Biological Chemistry, 1995
- Effect of chronic ethanol feeding on glutathione and functional integrity of mitochondria in periportal and perivenous rat hepatocytes.Journal of Clinical Investigation, 1994
- Rat KC cDNA cloning and mRNA expression in lung macrophages and fibroblastsBiochemical and Biophysical Research Communications, 1992
- Apoptosis in cultured rat hepatocytes: The effects of tumour necrosis factor α and interferon γThe Journal of Pathology, 1991
- Tumor necrosis factor Characterization at the molecular, cellular and in vivo levelFEBS Letters, 1991
- Impaired uptake of glutathione by hepatic mitochondria from chronic ethanol-fed rats. Tracer kinetic studies in vitro and in vivo and susceptibility to oxidant stress.Journal of Clinical Investigation, 1991
- Effect of chronic ethanol feeding on rat hepatocytic glutathione. Compartmentation, efflux, and response to incubation with ethanol.Journal of Clinical Investigation, 1987
- Ethanol-induced liver fibrosis in rats fed high fat dietHepatology, 1986
- Severe and Progressive Steatosis and Focal Necrosis in Rat Liver Induced by Continuous Intragastric Infusion of Ethanol and Low Fat DietHepatology, 1985