Nitric Oxide Ameliorates Actinomycin D/Endotoxin-Induced Apoptotic Liver Failure in Mice

This publication has 28 references indexed in Scilit:

• Tumor necrosis factor-induced hepatic DNA fragmentation as an early marker of T cell-dependent liver injury in mice
  Gastroenterology, 1995
• Altered responses to bacterial infection and endotoxic shock in mice lacking inducible nitric oxide synthase
  Cell, 1995
• Splenic B lymphocyte programmed cell death is prevented by nitric oxide release through mechanisms involving sustained Bcl-2 levels.
  Journal of Clinical Investigation, 1995
• The nitric oxide donors, azide and hydroxylamine, inhibit the programmed cell death of cytokine‐deprived human eosinophils
  FEBS Letters, 1995
• Nitric oxide produced by human B lymphocytes inhibits apoptosis and Epstein-Barr virus reactivation
  Cell, 1994
• NO at work
  Cell, 1994
• Activation of Soluble Guanylyl Cyclase by a Factor Other Than Nitric Oxide or Carbon Monoxide Contributes to the Vascular Hyporeactivity to Vasoconstrictor Agents in the Aorta of Rats Treated with Endotoxin
  Biochemical and Biophysical Research Communications, 1994
• A redox-based mechanism for the neuroprotective and neurodestructive effects of nitric oxide and related nitroso-compounds
  Nature, 1993
• Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis
  Nature, 1992
• Heat shock protects WEHI‐164 target cells from the cytolysis by tumor necrosis factors α and β
  European Journal of Immunology, 1989