Possible mechanisms of age-associated reduction of vascular relaxation caused by atrial natriuretic peptide

This publication has 15 references indexed in Scilit:

• L-arginine is the physiological precursor for the formation of nitric oxide in endothelium-dependent relaxation
  Biochemical and Biophysical Research Communications, 1988
• Age-related decrease in endothelium-dependent dilator response to histamine in rat mesenteric artery
  European Journal of Pharmacology, 1986
• Comparison of binding and cyclic GMP accumulation by atrial natriuretic peptides in endothelial cells
  Biochimica et Biophysica Acta (BBA) - Molecular Cell Research, 1986
• Effects of atrial natriuretic factor, sodium nitroprusside, and acetylcholine on cyclic GMP levels and relaxation in rat aorta
  European Journal of Pharmacology, 1985
• Atriopeptin II elevates cyclic GMP, activates cyclic GMP-dependent protein kinase and causes relaxation in rat thoracic aorta
  Biochimica et Biophysica Acta (BBA) - Molecular Cell Research, 1985
• Cyclic guanosine monophosphate mediates vascular relaxation induced by atrial natriuretic factor.
  Hypertension, 1985
• Distinct atrial natriuretic factor receptor sites on cultured bovine aortic smooth muscle and endothelial cells
  Biochemical and Biophysical Research Communications, 1985
• The increase of cGMP by atrial natriuretic factor correlates with the distribution of particulate guanylate cyclase
  FEBS Letters, 1985
• Agonist-induced endothelium-dependent relaxation in rat thoracic aorta may be mediated through cGMP.
  Circulation Research, 1983
• Methylene blue inhibits coronary arterial relaxation and guanylate cyclase activation by nitroglycerin, sodium nitrite, and amyl nitrite
  Canadian Journal of Physiology and Pharmacology, 1981