The etiology of acrylamide neuropathy: Enolase, phosphofructokinase, and glyceraldehyde-3-phosphate dehydrogenase activities in peripheral nerve, spinal cord, brain, and skeletal muscle of acrylamide-intoxicated cats
- 1 September 1981
- journal article
- research article
- Published by Elsevier in Toxicology and Applied Pharmacology
- Vol. 60 (2) , 324-333
- https://doi.org/10.1016/0041-008x(91)90235-7
Abstract
No abstract availableThis publication has 35 references indexed in Scilit:
- Axonal transport: a dynamic view of neuronal structuresTrends in Neurosciences, 1980
- MEASUREMENT OF NEURON‐SPECIFIC (NSE) AND NON‐NEURONAL (NNE) ISOENZYMES OF ENOLASE IN RAT, MONKEY AND HUMAN NERVOUS TISSUEJournal of Neurochemistry, 1979
- STUDIES ON THE BIOCHEMICAL BASIS OF DISTAL AXONOPATHIES–I. INHIBITION OF GLYCOLYSIS BY NEUROTOXIC HEXACARBON COMPOUNDSJournal of Neurochemistry, 1979
- FUNCTIONAL PROPERTIES OF NEURONAL AND GLIAL ISOENZYMES OF BRAIN ENOLASEJournal of Neurochemistry, 1978
- Unmyelinated nerve fibres in feline acrylamide neuropathyActa Neuropathologica, 1978
- Determination and characterization of neuron specific protein (NSP) associated enolase activityBiochemical and Biophysical Research Communications, 1976
- PHYSIOLOGICAL STUDIES OF THE DYING-BACK PHENOMENONBrain, 1975
- AXOPLASMIC FLOW IN AXONAL NEUROPATHIES I. AXOPLASMIC FLOW IN CATS WITH TOXIC NEUROPATHIESBrain, 1973
- FAST AXOPLASMIC TRANSPORT IN MAMMALIAN NERVE IN VITRO AFTER BLOCK OF GLYCOLYSIS WITH IODOACETIC ACIDJournal of Neurochemistry, 1971
- Fast Transport System of Materials in Mammalian Nerve FibersScience, 1969