Progression of cardiomyopathy and neuropathy after liver transplantation in a patient with familial amyloidotic polyneuropathy caused by tyrosine-77 transthyretin variant
Open Access
- 1 May 1999
- journal article
- case report
- Published by Wiley in Liver Transplantation and Surgery
- Vol. 5 (3) , 246-248
- https://doi.org/10.1002/lt.500050309
Abstract
No abstract availableKeywords
This publication has 9 references indexed in Scilit:
- EFFECT OF ORTHOTOPIC LIVER TRANSPLANTATION ON THE PROGRESSION OF FAMILIAL AMYLOIDOTIC POLYNEUROPATHYTransplantation, 1998
- LONG-TERM RESULTS OF LIVER TRANSPLANTATION IN FAMILIAL AMYLOIDOTIC POLYNEUROPATHY TYPE ITransplantation, 1997
- PROGRESSION OF VENTRICULAR WALL THICKENING AFTER LIVER TRANSPLANTATION FOR FAMILIAL AMYLOIDOSIS1Transplantation, 1997
- Clinical improvement and amyloid regression after liver transplantation in hereditary transthyretin amyloidosisThe Lancet, 1993
- Familial Amyloidosis: A Study of 52 North American-Born Patients Examined During a 30-Year PeriodMayo Clinic Proceedings, 1992
- Biochemical effect of liver transplantation in two Swedish patients with familial amyloidotic polyneuropathy (FAP‐met30)Clinical Genetics, 1991
- Role of variant prealbumin in the pathogenesis of familial amyloidotic polyneuropathy: Fate of normal and variant prealbumin in the circulationArchives of Biochemistry and Biophysics, 1989
- Biochemical marker in familial amyloidotic polyneuropathy, Portuguese type. Family studies on the transthyretin (prealbumin)-methionine-30 variant.Journal of Clinical Investigation, 1985
- A PECULIAR FORM OF PERIPHERAL NEUROPATHYBrain, 1952