TGF-β1 acts as a tumor suppressor of human malignant keratinocytes independently of Smad 4 expression and ligand-induced G1 arrest

This publication has 38 references indexed in Scilit:

• Novel inactivating mutations of transforming growth factor-? type I receptor gene in head-and-neck cancer metastases
  International Journal of Cancer, 2001
• Defective repression of c- myc in breast cancer cells: A loss at the core of the transforming growth factor β growth arrest program
  Proceedings of the National Academy of Sciences, 2001
• Role of Transforming Growth Factor-beta Signaling in Cancer
  JNCI Journal of the National Cancer Institute, 2000
• Transcriptional Activators of TGF-β Responses: Smads
  Cell, 1998
• Expression of a dominant negative type II TGF-β receptor in mouse skin results in an increase in carcinoma incidence and an acceleration of carcinoma development
  Oncogene, 1998
• Overexpression of autocrine TGF-β1 suppresses the growth of spindle epithelial cellsin vitro andin vivo in the rat 4NQO model of oral carcinogenesis
  International Journal of Cancer, 1997
• DPC4 (SMAD4) mediates transforming growth factor-β1 (TGF-β1) induced growth inhibition and transcriptional response in breast tumour cells
  Oncogene, 1997
• The absence of Harvey ras mutations during development and progression of squamous cell carcinomas of the head and neck
  British Journal of Cancer, 1993
• Loss of expression of transforming growth factor beta in skin and skin tumors is associated with hyperproliferation and a high risk for malignant conversion.
  Proceedings of the National Academy of Sciences, 1993
• Gene mutations and increased levels of p53 protein in human squamous cell carcinomas and their cell lines
  British Journal of Cancer, 1993