Inflammation in acute coronary syndromes.
- 1 August 1993
- journal article
- abstracts
- Published by Wolters Kluwer Health in Circulation
- Vol. 88 (2) , 800-803
- https://doi.org/10.1161/01.cir.88.2.800
Abstract
No abstract availableThis publication has 11 references indexed in Scilit:
- Increased expression of neutrophil and monocyte adhesion molecules in unstable coronary artery disease.Circulation, 1993
- Cyclic flow variations in a conscious dog model of coronary artery stenoses and endothelial injury correlate with acute ischemic heart disease syndromes in humansJournal of the American College of Cardiology, 1993
- Neutrophil induced oxidative injury of cardiac myocytes. A compartmented system requiring CD11b/CD18-ICAM-1 adherence.Journal of Clinical Investigation, 1992
- Leukocyte accumulation promoting fibrin deposition is mediated in vivo by P-selectin on adherent plateletsNature, 1992
- Frequency and severity of cyclic flow alternations and platelet aggregation predict the severity of neointimal proliferation following experimental coronary stenosis and endothelial injury.Proceedings of the National Academy of Sciences, 1991
- The neutrophil selectin LECAM-1 presents carbohydrate ligands to the vascular selectins ELAM-1 and GMP-140Cell, 1991
- Inflammation in the course of early myocardial ischemiaThe FASEB Journal, 1991
- Chemotactic factors regulate lectin adhesion molecule 1 (LECAM-1)-dependent neutrophil adhesion to cytokine-stimulated endothelial cells in vitro.Journal of Clinical Investigation, 1991
- Endothelial cell-associated platelet-activating factor: a novel mechanism for signaling intercellular adhesion.The Journal of cell biology, 1990
- Rapid neutrophil adhesion to activated endothelium mediated by GMP-140Nature, 1990