Lack of a C-terminal Tail in the Mammalian Gonadotropin-releasing Hormone Receptor Confers Resistance to Agonist-dependent Phosphorylation and Rapid Desensitization
Open Access
- 1 October 1999
- journal article
- Published by Elsevier
- Vol. 274 (42) , 30146-30153
- https://doi.org/10.1074/jbc.274.42.30146
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Evidence of an important and direct role for protein kinase C in agonist‐induced phosphorylation leading to desensitization of the angiotensin AT1A receptorBritish Journal of Pharmacology, 1997
- Stimulus-dependent Phosphorylation of G-protein-coupled Receptors by Casein Kinase 1αJournal of Biological Chemistry, 1997
- Endocytosis and recycling of G protein-coupled receptorsTrends in Pharmacological Sciences, 1997
- Epitope-tagged gonadotropin-releasing hormone receptors heterologously-expressed in mammalian (COS-1) and insect (Sf9) cells1Note: Supported by NIH Research Grant DK45519 to J.D.N.12Note: Presented as preliminary reports at the 1995 Endocrine Society Meetings (Washington, DC), and at the 1995 Society for Neuroscience Meetings (San Diego, CA).2Molecular and Cellular Endocrinology, 1997
- Desensitization of Gonadotropin-releasing Hormone Action in αT3-1 Cells Due to Uncoupling of Inositol 1,4,5-Trisphosphate Generation and Ca2+ MobilizationPublished by Elsevier ,1996
- Identification of the G Protein-coupled Receptor Kinase Phosphorylation Sites in the Human β2-Adrenergic ReceptorPublished by Elsevier ,1996
- A Single Mutation of the Neurokinin-2 (NK2) Receptor Prevents Agonist-induced DesensitizationJournal of Biological Chemistry, 1995
- Desensitization of cell signalling mediated by phosphoinositidase CTrends in Pharmacological Sciences, 1993
- Inositol trisphosphate and calcium signallingNature, 1993
- Removal of phosphorylation sites from the β2-adrenergic receptor delays onset of agonist-promoted desensitizationNature, 1988