Chronic nicotine exposure reduces N-methyl-d-aspartate receptor-mediated damage in the hippocampus without altering calcium accumulation or extrusion: evidence of calbindin-D28K overexpression
- 1 January 2001
- journal article
- Published by Elsevier in Neuroscience
- Vol. 102 (1) , 75-85
- https://doi.org/10.1016/s0306-4522(00)00450-4
Abstract
No abstract availableKeywords
This publication has 59 references indexed in Scilit:
- Comparison of the regional expression of nicotinic acetylcholine receptor ?7 mRNA and [125I]-?-bungarotoxin binding in human postmortem brainJournal of Comparative Neurology, 1997
- Mechanisms and Effects of Intracellular Calcium Buffering on Neuronal Survival in Organotypic Hippocampal Cultures Exposed to Anoxia/Aglycemia or to ExcitotoxinsJournal of Neuroscience, 1997
- Sources of variability in nicotine and cotinine levels with use of nicotine nasal spray, transdermal nicotine, and cigarette smokingBritish Journal of Clinical Pharmacology, 1997
- Minireview: Nicotinic Acetylcholine Receptors on Hippocampal Neurons: Distribution on the Neuronal Surface and Modulation of Receptor ActivityJournal of Receptors and Signal Transduction, 1997
- The Ability of Amyloid β‐Protein [AβP (1–40)] to Form Ca2+ Channels Provides a Mechanism for Neuronal Death in Alzheimer's DiseaseAnnals of the New York Academy of Sciences, 1994
- Nicotine-induced protection of cultured cortical neurons againstN-methyl-D-aspartate receptor-mediated glutamate cytotoxicityBrain Research, 1994
- Relationship between cigarette smoking and Alzheimer's disease in a population‐based case‐control studyNeurology, 1993
- Effects of nicotinic agonists on the NMDA receptorBrain Research, 1991
- Calbindin-D 28K in hippocampal organotypic culturesBrain Research, 1989
- Calcium transport and buffering in neuronsTrends in Neurosciences, 1988