Differentiation of F9 Teratocarcinoma Stem Cells to Primitive Endoderm Is Regulated by the Giα2/Gsα Axis via Phospholipase C and Not Adenylylcyclase

This publication has 20 references indexed in Scilit:

• Morphogen-induced Decline in G Triggers F9 Teratocarcinoma Stem Cell Progression via Phospholipase C and Mitogen-activated Protein Kinase
  Journal of Biological Chemistry, 1996
• Structural determinants for activation of the α-subunit of a heterotrimeric G protein
  Nature, 1994
• Regulation of the Differentiation of Teratocarcinoma Cells into Primitive Endoderm by Gβ _i2
  Science, 1992
• Retinoic acid regulates gene expression of retinoic acid receptors α,β and γ in F9 mouse teratocarcinoma cells
  Differentiation, 1992
• Identification of effector-activating residues of Gsα
  Cell, 1992
• Activating mutations in the NH₂‐ and COOH‐terminal moieties of the G_sα subunit have dominant phenotypes and distinguishable kinetics of adenylyl cyclase stimulation
  Journal of Cellular Biochemistry, 1991
• Gαi-αs chimeras define the function of α chain domains in control of G protein activation and βγ subunit complex interactions
  Cell, 1990
• Inositol trisphosphate receptor localization in brain: variable stoichiometry with protein kinase C
  Nature, 1987
• Interaction of heparin with plasminogen activators and plasminogen: effects on the activation of plasminogen
  Biochemistry, 1986
• The induction of differentiation in teratocarcinoma stem cells by retinoic acid
  Cell, 1978