Cytokine-induced apoptosis and necrosis are preceded by disruption of the mitochondrial membrane potential (Δψm) in pancreatic RINm5F cells: prevention by Bcl-2
- 1 April 2002
- journal article
- Published by Elsevier in Molecular and Cellular Endocrinology
- Vol. 190 (1-2) , 75-82
- https://doi.org/10.1016/s0303-7207(02)00009-6
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Reactive oxygen species (ROS) mediates the mitochondrial‐dependent apoptosis induced by transforming growth factor ß in fetal hepatocytesThe FASEB Journal, 2001
- Oxygen Reduction by Nitric-oxide SynthasesJournal of Biological Chemistry, 2001
- Bcl-2 prevents mitochondrial permeability transition and cytochrome c release via maintenance of reduced pyridine nucleotidesCell Death & Differentiation, 2000
- Suppression of Apoptosis by Nitric Oxide via Inhibition of Interleukin-1β–converting Enzyme (ICE)-like and Cysteine Protease Protein (CPP)-32–like ProteasesThe Journal of Experimental Medicine, 1997
- Mitochondrial control of apoptosisPublished by Elsevier ,1997
- Shift of the Cellular Oxidation‐Reduction Potential in Neural Cells Expressing Bcl‐2Journal of Neurochemistry, 1996
- Interleukin-1β Induced Activation of NF-κB in Insulin-Producing RINm5F Cells Is Prevented by the Protease Inhibitor Nα-p-Tosyl-L-Lysine ChloromethylketoneBiochemical and Biophysical Research Communications, 1994
- Peroxynitrite causes calcium efflux from mitochondria which is prevented by Cyclosporin AFEBS Letters, 1994
- Metabolism and β-cell function of rat pancreatic islets exposed to human interleukin-1β in the presence of a high glucose concentrationImmunology Letters, 1990
- Establishment of mouse cell lines which constitutively secrete large quantities of interleukin 2, 3, 4 or 5, using modified cDNA expression vectorsEuropean Journal of Immunology, 1988