Protein Kinase Cθ Is Required for Autophagy in Response to Stress in the Endoplasmic Reticulum
Open Access
- 1 May 2008
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 283 (22) , 15370-15380
- https://doi.org/10.1074/jbc.m710209200
Abstract
No abstract availableKeywords
This publication has 66 references indexed in Scilit:
- Autophagy fights disease through cellular self-digestionNature, 2008
- Autophagy in the Pathogenesis of DiseaseCell, 2008
- Signal integration in the endoplasmic reticulum unfolded protein responseNature Reviews Molecular Cell Biology, 2007
- Control of Macroautophagy by Calcium, Calmodulin-Dependent Kinase Kinase-β, and Bcl-2Molecular Cell, 2007
- Autophagy Is Activated for Cell Survival after Endoplasmic ReticulumStressMolecular and Cellular Biology, 2006
- Autophagy Counterbalances Endoplasmic Reticulum Expansion during the Unfolded Protein ResponsePLoS Biology, 2006
- Adaptation to ER Stress Is Mediated by Differential Stabilities of Pro-Survival and Pro-Apoptotic mRNAs and ProteinsPLoS Biology, 2006
- Endoplasmic Reticulum Stress Triggers AutophagyJournal of Biological Chemistry, 2006
- PKCθ is a key player in the development of insulin resistanceBiochemical and Biophysical Research Communications, 2006
- Autophagy: molecular machinery for self-eatingCell Death & Differentiation, 2005