Deficient transforming growth factor β and interleukin-10 responses contribute to the septic death of burned patients
- 1 November 2002
- Vol. 28 (7) , 631-637
- https://doi.org/10.1016/s0305-4179(02)00113-4
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Impact of Burn Injury on Hepatic TGF-??1 Expression and Plasma TGF-??1 LevelsPublished by Wolters Kluwer Health ,2000
- THERAPEUTIC IMPLICATIONS OF INTERLEUKIN-10 IN SURGICAL DISEASEShock, 1998
- Termination of Acute-Phase ResponseGeneral Pharmacology: The Vascular System, 1998
- Changes in serum tumour necrosis factor-α in burned patientsBurns, 1997
- Pathophysiology of the burn wound and pharmacological treatment. The Rudi Hermans Lecture, 1995Burns, 1996
- Interleukin 10 reduces the release of tumor necrosis factor and prevents lethality in experimental endotoxemia.The Journal of Experimental Medicine, 1993
- Targeted disruption of the mouse transforming growth factor-β1 gene results in multifocal inflammatory diseaseNature, 1992
- Induction of the interleukin 1 receptor antagonist protein by transforming growth factor‐βEuropean Journal of Immunology, 1991
- Deactivation of macrophages by transforming growth factor-βNature, 1988
- Inhibition of cytokine production by cyclosporin A and transforming growth factor beta.The Journal of Experimental Medicine, 1987