Trapping of growth factors by catechins: a possible therapeutical target for prevention of proliferative diseases
- 26 April 2005
- journal article
- review article
- Published by Elsevier in The Journal of Nutritional Biochemistry
- Vol. 16 (5) , 259-266
- https://doi.org/10.1016/j.jnutbio.2004.11.003
Abstract
No abstract availableKeywords
This publication has 60 references indexed in Scilit:
- Epigallocatechin-3-gallate, constituent of green tea, suppresses the LPS-induced phenotypic and functional maturation of murine dendritic cells through inhibition of mitogen-activated protein kinases and NF-κBBiochemical and Biophysical Research Communications, 2003
- Treatment of epigallocatechin‐3‐gallate inhibits matrix metalloproteinases‐2 and ‐9 via inhibition of activation of mitogen‐activated protein kinases, c‐jun and NF‐κB in human prostate carcinoma DU‐145 cellsThe Prostate, 2003
- The biology of VEGF and its receptorsNature Medicine, 2003
- A Major Constituent of Green Tea, EGCG, Inhibits the Growth of a Human Cervical Cancer Cell Line, CaSki Cells, through Apoptosis, G1 Arrest, and Regulation of Gene ExpressionDNA and Cell Biology, 2003
- ‘Angioprevention’: angiogenesis is a common and key target for cancer chemopreventive agentsThe FASEB Journal, 2002
- Natural antimutagenic agents may prolong efficacy of human immunodeficiency virus drug therapyMedical Hypotheses, 1997
- Highlights of the Cancer Chemoprevention Studies in ChinaPreventive Medicine, 1993
- Inhibitory effect of green tea on tumorigenesis by chemicals and ultraviolet lightPreventive Medicine, 1992
- (—)‐Epigallocatechin‐3‐gallate incamellia sinensisleaves from Himalayan region of Sikkim: Inhibitory effects against biochemical events and tumor initiation in sencar mouse skinNutrition and Cancer, 1992
- Crude tea extracts decrease the mutagenic activity of N-methyl-N′-nitro-N-nitrosoguanidine in vitro and in intragastric tract of ratsMutation Research - Fundamental and Molecular Mechanisms of Mutagenesis, 1989