Decreased protein synthetic activity of the hypothalamic tuberomamillary nucleus in Alzheimer's disease as suggested by smaller Golgi apparatus
- 23 June 1995
- journal article
- Published by Elsevier in Neuroscience Letters
- Vol. 193 (1) , 29-32
- https://doi.org/10.1016/0304-3940(95)11659-k
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- Early cytoskeletal changes as shown by Alz-50 are not accompanied by decreased neuronal activityBrain Research, 1995
- The Golgi apparatus of moptor neurons in amyotrophic lateral sclerosisAnnals of Neurology, 1993
- The distribution of Alz-50 immunoreactivity in the hypothalamus and adjoining areas of Alzheimer's disease patientsBrain, 1993
- Brain aging and Alzheimer's disease, “Wear and tear” versus “Use it or lose it”Neurobiology of Aging, 1991
- Alz-50 immunoreactivity in the hypothalamus of the normal and Alzheimer human and the ratJournal of Comparative Neurology, 1991
- Immunocytochemical visualization of the Golgi apparatus in several species, including human, and tissues with an antiserum against MG-160, a sialoglycoprotein of rat Golgi apparatus.Journal of Histochemistry & Cytochemistry, 1990
- Hypothalamic pathology in Alzheimer's diseaseNeuroscience Letters, 1987
- The precursor of Alzheimer's disease amyloid A4 protein resembles a cell-surface receptorNature, 1987
- Clinical diagnosis of Alzheimer's diseaseNeurology, 1984
- The Golgi apparatus (complex)-(1954-1981)-from artifact to center stage.The Journal of cell biology, 1981