Angiotensin II and guanine nucleotides stimulate formation of inositol 1,4,5-trisphosphate and its metabolites in permeabilized adrenal glomerulosa cells.

This publication has 42 references indexed in Scilit:

• Signal transduction by guanine nucleotide binding proteins
  Molecular and Cellular Endocrinology, 1987
• The role of guanyl nucleotide binding proteins in the formation of inositol phosphates in adrenal glomerulosa cells
  Biochemical and Biophysical Research Communications, 1986
• Pertussis toxin blocks angiotensin II‐induced calcium influx but not inositol trisphosphate production in adrenal glomerulosa cell
  FEBS Letters, 1986
• Direct stimulation by thyrotropin-releasing hormone (TRH) of polyphosphoinositide hydrolysis in GH3 cell membranes by a guanine nucleotide-modulated mechanism
  Biochemical and Biophysical Research Communications, 1985
• Role of guanine nucleotide binding protein in the activation of polyphosphoinositide phosphodiesterase
  Nature, 1985
• Inositol trisphosphate, a novel second messenger in cellular signal transduction
  Nature, 1984
• Guanine nucleotides decrease the free [Ca²⁺] required for secretion of serotonin from permeabilized blood platelets
  FEBS Letters, 1984
• Receptor-Induced Diacylglycerol Formation in Permeabilized Platelets; Possible Role for a Gtp-Binding Protein
  Journal of Receptor Research, 1984
• Islet-activating protein, pertussis toxin: a probe for functions of the inhibitory guanine nucleotide regulatory component of adenylate cyclase
  Trends in Pharmacological Sciences, 1984
• Calcium-dependence of catecholamine release from bovine adrenal medullary cells after exposure to intense electric fields
  The Journal of Membrane Biology, 1982