Complexity and Redundancy in the Pathogenesis of Asthma: Reassessing the Roles of Mast Cells and T Cells

This publication has 42 references indexed in Scilit:

• Aeroallergen-induced eosinophilic inflammation, lung damage, and airways hyperreactivity in mice can occur independently of IL-4 and allergen-specific immunoglobulins.
  Journal of Clinical Investigation, 1997
• Central role of immunoglobulin (Ig) E in the induction of lung eosinophil infiltration and T helper 2 cell cytokine production: inhibition by a non-anaphylactogenic anti-IgE antibody.
  The Journal of Experimental Medicine, 1996
• Interleukin 4, but not interleukin 5 or eosinophils, is required in a murine model of acute airway hyperreactivity.
  The Journal of Experimental Medicine, 1996
• Sorting out the cytokines of asthma.
  The Journal of Experimental Medicine, 1996
• The consequences of chronic allergic inflammation.
  Thorax, 1995
• Mast cell activation enhances airway responsiveness to methacholine in the mouse.
  Journal of Clinical Investigation, 1993
• Predominant T_H2-like Bronchoalveolar T-Lymphocyte Population in Atopic Asthma
  New England Journal of Medicine, 1992
• Differences in the expression of the cardiopulmonary alterations associated with anti-immunoglobulin E-induced or active anaphylaxis in mast cell-deficient and normal mice. Mast cells are not required for the cardiopulmonary changes associated with certain fatal anaphylactic responses.
  Journal of Clinical Investigation, 1991
• The Immunobiology of Eosinophils
  New England Journal of Medicine, 1991
• Fate of bone marrow-derived cultured mast cells after intracutaneous, intraperitoneal, and intravenous transfer into genetically mast cell-deficient W/Wv mice. Evidence that cultured mast cells can give rise to both connective tissue type and mucosal mast cells.
  The Journal of Experimental Medicine, 1985