Abnormal KCNQ1 trafficking influences disease pathogenesis in hereditary long QT syndromes (LQT1)
Open Access
- 15 August 2005
- journal article
- Published by Oxford University Press (OUP) in Cardiovascular Research
- Vol. 67 (3) , 476-486
- https://doi.org/10.1016/j.cardiores.2005.04.036
Abstract
Objective: In the hereditary long QT syndromes the commonest defect is in the K+ channel pore forming subunit, KCNQ1. In this study we investigated tKeywords
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