Expression of the c-myb proto-oncogene during cellular proliferation
- 1 January 1986
- journal article
- research article
- Published by Springer Nature in Nature
- Vol. 319 (6052) , 374-380
- https://doi.org/10.1038/319374a0
Abstract
In several cell types, messenger RNA levels of the nuclear proto-oncogene c-myb vary as a function of cellular proliferation; a transient increase in c-myb steady-state mRNA, mediated by post-transcriptional mechanisms, occurs during cell-cycle progression. In contrast, both quiescent and proliferating immature thymocytes contain exceptionally high levels of c-myb mRNA as a consequence of increased c-myb transcription.This publication has 36 references indexed in Scilit:
- Levels of c-myc oncogene mRNA are invariant throughout the cell cycleNature, 1985
- V-myc- and c-myc-encoded proteins are associated with the nuclear matrix.Molecular and Cellular Biology, 1985
- Cell cycle-dependent expression of thymidylate synthase in Saccharomyces cerevisiae.Molecular and Cellular Biology, 1984
- Subcellular localization of proteins encoded by oncogenes of avian myeloblastosis virus and avian leukemia virus E26 and by the chicken c-myb geneCell, 1984
- Growth factors: Mechanism of action and relation to oncogenesCell, 1984
- Post-transcriptional regulation of the chicken thymidine kinase geneNucleic Acids Research, 1984
- Viral and cellular fos proteins: A comparative analysisCell, 1984
- Control of dihydrofolate reductase messenger ribonucleic acid production.Molecular and Cellular Biology, 1981
- The nature of conditionally lethal temperature-sensitive mutations in somatic cellsJournal of Cellular Physiology, 1978
- Genetic Control of the Cell Division Cycle in YeastScience, 1974