Neuronal MeCP2 Is Expressed at Near Histone-Octamer Levels and Globally Alters the Chromatin State
Top Cited Papers
- 1 February 2010
- journal article
- Published by Elsevier in Molecular Cell
- Vol. 37 (4) , 457-468
- https://doi.org/10.1016/j.molcel.2010.01.030
Abstract
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This publication has 47 references indexed in Scilit:
- Phosphorylation of MeCP2 at Serine 80 regulates its chromatin association and neurological functionProceedings of the National Academy of Sciences, 2009
- Non–cell autonomous influence of MeCP2-deficient glia on neuronal dendritic morphologyNature Neuroscience, 2009
- MeCP2, a Key Contributor to Neurological Disease, Activates and Represses TranscriptionScience, 2008
- Integrated epigenomic analyses of neuronal MeCP2 reveal a role for long-range interaction with active genesProceedings of the National Academy of Sciences, 2007
- Cerebellar gene expression profiles of mouse models for Rett syndrome reveal novel MeCP2 targetsBMC Medical Genetics, 2007
- Multiple Modes of Interaction between the Methylated DNA Binding Protein MeCP2 and ChromatinMolecular and Cellular Biology, 2007
- MeCP2 Behaves as an Elongated Monomer That Does Not Stably Associate with the Sin3a Chromatin Remodeling ComplexPublished by Elsevier ,2004
- Deletions of a differentially methylated CpG island at the SNRPN gene define a putative imprinting control regionNature Genetics, 1994
- Purification, sequence, and cellular localization of a novel chromosomal protein that binds to Methylated DNACell, 1992
- A fraction of the mouse genome that is derived from islands of nonmethylated, CpG-rich DNAPublished by Elsevier ,1985