Thalamic mediodorsal and intralaminar nuclear lesions disrupt the generation of experimentally induced generalized absence-like seizures in rats
- 17 March 1994
- journal article
- Published by Elsevier in Epilepsy Research
- Vol. 17 (3) , 193-205
- https://doi.org/10.1016/0920-1211(94)90050-7
Abstract
No abstract availableKeywords
This publication has 25 references indexed in Scilit:
- γ-Hydroxybutyric acid induced spike and wave discharges in rats: Relation to high-affinity [3H]γ-hydroxybutyric acid binding sites in the thalamus and cortexNeuroscience, 1993
- Suppressive effects of intranigral injection of muscimol in three models of generalized non-convulsive epilepsy induced by chemical agentsBrain Research, 1989
- The THIP-induced model of bilateral synchronous spike and wave in rodentsNeuropharmacology, 1987
- The role of GABA and excitatory amino acids in the development of the leptazol-induced epileptogenic EEGNeuropharmacology, 1986
- Role of the thalamus in generalized penicillin epilepsy: Observations on decorticated catsExperimental Neurology, 1982
- Interaction of cortex and thalamus in spike and wave discharges of feline generalized penicillin epilepsyExperimental Neurology, 1982
- Laboratory noteElectroencephalography and Clinical Neurophysiology, 1980
- Laminar Organization of Thalamic Projections to the Rat NeocortexScience, 1980
- Generalized Epilepsy with Spike‐and‐Wave Discharge: A Reinterpretation of Its Electrographic and Clinical Manifestations1Epilepsia, 1979
- Cortical-thalamic relationships in the ratExperimental Neurology, 1975