Abolishing Bax-Dependent Apoptosis Shows Beneficial Effects on Spinal Muscular Atrophy Model Mice
Open Access
- 30 June 2006
- journal article
- Published by Elsevier in Molecular Therapy
- Vol. 13 (6) , 1149-1155
- https://doi.org/10.1016/j.ymthe.2006.02.008
Abstract
No abstract availableKeywords
This publication has 25 references indexed in Scilit:
- Upregulation of Bax protein and increased DNA degradation in ALS spinal cord motor neuronsActa Neurologica Scandinavica, 2009
- An update of the mutation spectrum of the survival motor neuron gene (SMN1) in autosomal recessive spinal muscular atrophy (SMA)Human Mutation, 2000
- A mouse model for spinal muscular atrophyNature Genetics, 2000
- Bax and Bcl‐2 Interaction in a Transgenic Mouse Model of Familial Amyotrophic Lateral SclerosisJournal of Neurochemistry, 1999
- Widespread Elimination of Naturally Occurring Neuronal Death inBax-Deficient MiceJournal of Neuroscience, 1998
- The role of apoptosis in neurodegenerative diseases.Metabolic Brain Disease, 1998
- BCL-2 GENE FAMILY IN THE NERVOUS SYSTEMAnnual Review of Neuroscience, 1997
- BAX Is Required for Neuronal Death after Trophic Factor Deprivation and during DevelopmentNeuron, 1996
- International SMA Consortium Meeting (26–28 June 1992, Bonn, Germany)Neuromuscular Disorders, 1992
- Incidence, prevalence, and gene frequency studies of chronic childhood spinal muscular atrophy.Journal of Medical Genetics, 1978