Abstract
Phosphorylation of the 18,000-dalton L chains of the fast-twitch myosin in mouse extensor digitorum longus muscles was correlated with reduction in the rate of the actomyosin ATPase in vivo, but neither of these changes occurred in the soleus muscle. Apparently actomyosin interactions can be down-regulated by a reversible covalent modification of myosin L chains; a mechanism for thick-filament regulation occurs in vertebrate skeletal muscle; and the expression of this regulation may be limited to a specific fiber type.

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