Chapter 18 Plasticity of the nervous system at the systemic, cellular and molecular levels: a mechanism of chronic pain and hyperalgesia
- 1 January 1996
- book chapter
- Published by Elsevier
- Vol. 110, 233-259
- https://doi.org/10.1016/s0079-6123(08)62578-9
Abstract
No abstract availableThis publication has 88 references indexed in Scilit:
- Inhibition of carrageenan-induced spinal c-Fos activation by systemically administered c-fos antisense oligodeoxynucleotides may be facilitated by local opening of the blood-spinal cord barrierJournal of Neuroscience Research, 1997
- Comparison of c‐jun, junB, and junD mRNA expression and protein in the rat dorsal root ganglia following sciatic nerve transectionJournal of Neuroscience Research, 1995
- Gap‐43 immunoreactivity and axon regeneration in retinal ganglion cells of the ratJournal of Neurobiology, 1994
- Inhibition of c‐Fos Protein Expression in Rat Spinal Cord by Antisense Oligodeoxynucleotide SuperfusionEuropean Journal of Neuroscience, 1994
- Expression of JUN, KROX, and CREB transcription factors in goldfish and rat retinal ganglion cells following optic nerve lesion is related to axonal sproutingJournal of Neurobiology, 1993
- Sequential expression of JUN B, JUN D and FOS B proteins in rat spinal neurons: Cascade of transcriptional operations during nociceptionNeuroscience Letters, 1991
- The sensory-efferent function of capsaicin-sensitive sensory neuronsGeneral Pharmacology: The Vascular System, 1988
- Neuropeptides in physiologically identified mammalian sensory neuronesNeuroscience Letters, 1985
- Effects of capsaicin on inflammation and on the substance P content of nervous tissues in rats with adjuvant arthritisLife Sciences, 1983
- Deafferentation hypersensitivity in the rat after dorsal rhizotomy: A possible animal model of chronic painPain, 1979