Cardiac overexpression of antioxidant catalase attenuates aging-induced cardiomyocyte relaxation dysfunction
- 31 March 2007
- journal article
- Published by Elsevier in Mechanisms of Ageing and Development
- Vol. 128 (3) , 276-285
- https://doi.org/10.1016/j.mad.2006.12.007
Abstract
No abstract availableKeywords
This publication has 41 references indexed in Scilit:
- Metallothionein prolongs survival and antagonizes senescence‐associated cardiomyocyte diastolic dysfunction: role of oxidative stressThe FASEB Journal, 2006
- Oxidative Stress and Aging: Catalase Is a Longevity Determinant EnzymeRejuvenation Research, 2005
- Aging induces cardiac diastolic dysfunction, oxidative stress, accumulation of advanced glycation endproducts and protein modificationAging Cell, 2005
- Age-dependent suppression of SERCA2a mRNA in pediatric atrial myocardiumBiochemical and Biophysical Research Communications, 2005
- Mechanisms underlying enhanced cardiac excitation contraction coupling observed in the senescent sheep myocardiumJournal of Molecular and Cellular Cardiology, 2004
- Cardiac-specific overexpression of catalase rescues ventricular myocytes from ethanol-induced cardiac contractile defectJournal of Molecular and Cellular Cardiology, 2003
- Alterations in Properties of L-Type Ca Channels in Aging Rat HeartJournal of Molecular and Cellular Cardiology, 2002
- Ischemia-reperfusion in the adult mouse heartExperimental Gerontology, 1999
- Expressional analysis of the cardiac Na–Ca exchanger in rat development and senescenceCardiovascular Research, 1998
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976