The effect of acute inflammation on iron metabolism in rats.

Abstract
Fe metabolism in rats with acute turpentine-induced inflammation was evaluated. In acute inflammation, reduced plasma Fe and total Fe-binding capacity values, shortened plasma Fe disappearance time and lower plasma Fe turnover were observed. Administration of 59Fe chondroitin ferrous sulfate to evaluate RES function revealed a significantly increased 59Fe retention in the liver and lower incorporation into red blood cells. Radioactivity in hepatic RE cells was higher in acute inflammation than in control. A block in the transfer of Fe from RES cells to the plasma Fe pool may occur during acute inflammation.

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