Effect of the transcriptional repressor Mad1 on proliferation of human melanoma cells
- 4 October 2002
- journal article
- research article
- Published by Wiley in Experimental Dermatology
- Vol. 11 (5) , 439-447
- https://doi.org/10.1034/j.1600-0625.2002.110507.x
Abstract
Mad1 is a Myc antagonist that heterodimerizes with Max and functions as a transcriptional repressor. We studied the effects of Mad1 on cell growth and malignant phenotype in human melanoma ...Keywords
This publication has 26 references indexed in Scilit:
- c-MYC and nodular malignant melanomaCancer, 2000
- Measurement of c-myc oncoprotein provides an independent prognostic marker for regional metastatic melanomaBritish Journal of Plastic Surgery, 1997
- The clinical significance of oncogene expression in subungual melanomaBritish Journal of Plastic Surgery, 1997
- C-myc oncogene expression in human melanoma and its relationship with tumour antigenicityEuropean Journal of Surgical Oncology, 1996
- Suppression of the Malignant Phenotype of Melanoma Cells by Anti-Oncogene RibozymesJournal of Investigative Dermatology, 1996
- Contrasting roles for Myc and Mad proteins in cellular growth and differentiation.Proceedings of the National Academy of Sciences, 1995
- Immunohistochemical expression of C-myc oncogene, heat shock protein 70 and HLA-DR molecules in malignant cutaneous melanomaVirchows Archiv, 1995
- Myc—Max—Mad: a transcription factor network controlling cell cycle progression, differentiation and deathCurrent Opinion in Genetics & Development, 1994
- Mad: A heterodimeric partner for Max that antagonizes Myc transcriptional activityCell, 1993
- Cutaneous MelanomaNew England Journal of Medicine, 1991