Tissue plasminogen activator and plasmin independently decrease human neutrophil activation
- 1 January 1989
- journal article
- research article
- Published by Elsevier in Life Sciences
- Vol. 45 (18) , 1665-1669
- https://doi.org/10.1016/0024-3205(89)90276-2
Abstract
No abstract availableThis publication has 22 references indexed in Scilit:
- Neutrophil function in ischemic heart disease.Circulation, 1989
- Impaired canine coronary vasodilator response to acetylcholine and bradykinin after occlusion-reperfusion.Circulation Research, 1989
- Oxygen-derived free radicals and postischemic myocardial dysfunction (“stunned myocardium”)Journal of the American College of Cardiology, 1988
- Protection of Myocardial Function and Coronary Vasculature by StreptokinaseJournal of Cardiovascular Pharmacology, 1988
- Streptokinase-induced, antibody-mediated platelet aggregation: A potential cause of clot propagation in vivoJournal of the American College of Cardiology, 1988
- Neutrophils as potential participants in acute myocardial ischemia: Relevance to reperfusionJournal of the American College of Cardiology, 1988
- Modulation of human neutrophil superoxide production by selective thromboxane synthetase inhibitor U63, 557ALife Sciences, 1988
- Tissue plasminogen activator promotes platelet disaggregation in plasma.Journal of Clinical Investigation, 1987
- Effects of endogenously produced leukotrienes, thromboxane, and prostaglandins on coronary vascular resistance in rabbit myocardial infarction.Journal of Clinical Investigation, 1985
- Nonplasmin-Mediated FibrinolysisSeminars in Thrombosis and Hemostasis, 1984