Omeprazole, a specific inhibitor of gastric (H+-K+)-ATPase, is a H+-activated oxidizing agent of sulfhydryl groups.
Open Access
- 1 April 1985
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 260 (8) , 4591-4597
- https://doi.org/10.1016/s0021-9258(18)89112-8
Abstract
No abstract availableKeywords
This publication has 21 references indexed in Scilit:
- Effect of carbachol or histamine stimulation on rat gastric membranes enriched in (H+-K+)-ATPaseBiochimica et Biophysica Acta (BBA) - Biomembranes, 1984
- Kinetic properties of the KCl transport at the secreting apical membrane of the oxyntic cellThe Journal of Membrane Biology, 1983
- Inhibition of gastric (H+ + K+)-ATPase by the substituted benzimidazole, picoprazoleBiochimica et Biophysica Acta (BBA) - Biomembranes, 1983
- Inhibition of gastric by unsaturated long-chain fatty acidsBiochimica et Biophysica Acta (BBA) - Biomembranes, 1982
- Substituted benzimidazoles inhibit gastric acid secretion by blocking (H+ + K+) ATPaseNature, 1981
- Mechanisms of Gastric H+ and C1- TransportAnnual Review of Physiology, 1980
- Characterization of gastric mucosal membranes. X. Immunological studies of gastric (H+ + K+)-ATPase.The Journal of cell biology, 1979
- Quantitation of hydrogen ion and potential gradients in gastric plasma membrane vesiclesBiochemistry, 1978
- Isolation and characterization of the plasma membrane of L-1210 cells. Iodination as a marker for the plasma membraneBiochimica et Biophysica Acta (BBA) - Biomembranes, 1975
- Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4Nature, 1970