Pathobiologic Responses to Stenting 11Supported in part by grants from the National Institutes of Health (GM/HL49039 and HL03104), the Burroughs Wellcome Fund for Experimental Therapeutics, Durham, North Carolina, and the Whitaker Foundation, Rosslyn, Virginia.

This publication has 11 references indexed in Scilit:

• Experimental Study of Thrombogenicity and Foreign Body Reaction Induced by Heparin-Coated Coronary Stents
  Circulation, 1997
• Monocyte Recruitment and Neointimal Hyperplasia in Rabbits
  Arteriosclerosis, Thrombosis, and Vascular Biology, 1996
• Morphologic characteristics of lesion formation and time course of smooth muscle cell proliferation in a porcine proliferative restenosis model
  Journal of the American College of Cardiology, 1994
• A Comparison of Balloon-Expandable-Stent Implantation with Balloon Angioplasty in Patients with Coronary Artery Disease
  New England Journal of Medicine, 1994
• A Randomized Comparison of Coronary-Stent Placement and Balloon Angioplasty in the Treatment of Coronary Artery Disease
  New England Journal of Medicine, 1994
• Inhibition of experimental neointimal hyperplasia and thrombosis depends on the type of vascular injury and the site of drug administration.
  Circulation, 1993
• Coronary intimal proliferation after balloon injury and stenting in swing: An animal model of restenosis
  Journal of the American College of Cardiology, 1992
• Restenosis and the proportional neointimal response to coronary artery injury: Results in a porcine model
  Journal of the American College of Cardiology, 1992
• Incidence of restenosis after successful coronary angioplasty: a time-related phenomenon. A quantitative angiographic study in 342 consecutive patients at 1, 2, 3, and 4 months.
  Circulation, 1988
• Intravascular Stents to Prevent Occlusion and Re-Stenosis after Transluminal Angioplasty
  New England Journal of Medicine, 1987