Why should an angiogenic factor modulate tubular structure in diabetic nephropathy? Some answers, more questions
- 1 August 2003
- journal article
- Published by Elsevier in Kidney International
- Vol. 64 (2) , 758-759
- https://doi.org/10.1046/j.1523-1755.2003.00142.x
Abstract
No abstract availableKeywords
This publication has 11 references indexed in Scilit:
- Vascular endothelial growth factor induces protein synthesis in renal epithelial cells: A potential role in diabetic nephropathy11See Editorial by Ziyadeh and Wolf, p. 758.Kidney International, 2003
- Neutralization of Circulating Vascular Endothelial Growth Factor (VEGF) by Anti-VEGF Antibodies and Soluble VEGF Receptor 1 (sFlt-1) Induces ProteinuriaJournal of Biological Chemistry, 2003
- Amelioration of Long-Term Renal Changes in Obese Type 2 Diabetic Mice by a Neutralizing Vascular Endothelial Growth Factor AntibodyDiabetes, 2002
- Effects of high glucose and TGF-β1 on the expression of collagen IV and vascular endothelial growth factor in mouse podocytesKidney International, 2002
- Vascular Endothelial Growth Factor Enhances Glomerular Capillary Repair and Accelerates Resolution of Experimentally Induced GlomerulonephritisThe American Journal of Pathology, 2001
- Ornithine decarboxylase, kidney size, and the tubular hypothesis of glomerular hyperfiltration in experimental diabetesJournal of Clinical Investigation, 2001
- Hypoxia and interleukin-1β stimulate vascular endothelial growth factor production in human proximal tubular cellsKidney International, 2000
- Increased renal expression of vascular endothelial growth factor (VEGF) and its receptor VEGFR-2 in experimental diabetes.Diabetes, 1999
- Molecular mechanisms of diabetic renal hypertrophyKidney International, 1999
- Vascular endothelial growth factor (VEGF) and its receptorsThe FASEB Journal, 1999