Pathophysiology of ANCA-associated vasculitides: Are ANCA really pathogenic?
- 1 May 2004
- journal article
- review article
- Published by Elsevier in Kidney International
- Vol. 65 (5) , 1564-1567
- https://doi.org/10.1111/j.1523-1755.2004.05412.x
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- SCG/Kinjoh mice: A model of ANCA-associated crescentic glomerulonephritis with immune depositsKidney International, 2003
- Expression profile of leukocyte genes activated by anti-neutrophil cytoplasmic autoantibodies (ANCA)Kidney International, 2002
- Antineutrophil cytoplasm autoantibodies from patients with systemic vasculitis activate neutrophils through distinct signaling cascades: comparison with conventional Fcγ receptor ligationBlood, 2001
- Priming of the Neutrophil Respiratory Burst Involves p38 Mitogen-activated Protein Kinase-dependent Exocytosis of Flavocytochrome b 558-containing GranulesJournal of Biological Chemistry, 2000
- Priming differentially regulates neutrophil adhesion molecule expression/functionImmunology, 1996
- Differences between anti-myeloperoxidase- and anti-proteinase 3-associated renal diseaseKidney International, 1995
- Antimyeloperoxidase Antibodies Induce Neutrophil Adherence to Cultured Human Endothelial CellsRenal Failure, 1995
- Activation of granulocytes by anti-neutrophil cytoplasmic antibodies (ANCA): a FcγRII-dependent processClinical and Experimental Immunology, 1994
- Activated neutrophils express proteinase 3 on their plasma membrane in vitro and in vivoClinical and Experimental Immunology, 1994
- Antimyeloperoxidase-associated proliferative glomerulonephritis: an animal model.The Journal of Experimental Medicine, 1993