TNF‐α increases ubiquitin‐conjugating activity in skeletal muscle by up‐regulating UbcH2/E220k

Abstract
In some inflammatory diseases, TNF-α is thought to stimulate muscle catabolism via an NF-κB-dependent process that increases ubiquitin conjugation to muscle proteins. The transcriptional mechanism of...
Funding Information
  • National Institutes of Health (DK02707, HL59878)
  • National Space Biomedical Research Institute
  • Muscular Dystrophy Association