Prostaglandins inhibit pancreatic β-cell replication and long-term insulin secretion by pertussis toxin-insensitive mechanisms but do not mediate the actions of interleukin-1 β
- 28 August 1996
- journal article
- Published by Elsevier in Biochimica et Biophysica Acta (BBA) - Molecular Cell Research
- Vol. 1313 (2) , 106-110
- https://doi.org/10.1016/0167-4889(96)00058-4
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- Intraislet release of interleukin 1 inhibits beta cell function by inducing beta cell expression of inducible nitric oxide synthase.The Journal of Experimental Medicine, 1995
- IL-1.beta. induces the coexpression of both nitric oxide synthase and cyclooxygenase by Islets of Langerhans: activation of cyclooxygenase by nitric oxideBiochemistry, 1993
- Arachidonic acid in cell signalingCurrent Opinion in Cell Biology, 1993
- Intracellular signal transduction pathways that control pancreatic β‐cell proliferationFEBS Letters, 1992
- Inhibition of fetal rat pancreatic β‐cell replication by interleukin‐1β in vitro is not mediated through pertussis toxin‐sensitive G‐proteins, a decrease in cyclic AMP, or protease activationFEBS Letters, 1991
- Effects of dexamethasone on glucose-induced insulin and proinsulin release in low and high insulin respondersMetabolism, 1990
- A natural interleukin 1 (IL-1) inhibitor counteracts the inhibitory effect of IL-1 on insulin production in cultured rat pancreatic isletsJournal of Autoimmunity, 1989
- Immune Hormones (Cytokines); Pathogenic Role in Autoimmune Rheumatic and Endocrine DiseasesAutoimmunity, 1989
- Type I Diabetes MellitusNew England Journal of Medicine, 1986
- Prostacyclin Synthesis Induced in Vascular Cells by Interleukin-1Science, 1985