Guanine nucleotide binding proteins and cellular control
- 1 April 1989
- journal article
- review article
- Published by Elsevier in Current Opinion in Cell Biology
- Vol. 1 (2) , 196-200
- https://doi.org/10.1016/0955-0674(89)90086-0
Abstract
No abstract availableThis publication has 21 references indexed in Scilit:
- Purification of heterotrimeric GTP-binding proteins from brain: identification of a novel form of GoBiochemistry, 1988
- Site of G Protein Binding to Rhodopsin Mapped with Synthetic Peptides from the α SubunitScience, 1988
- The stimulatory G protein of adenylyl cyclase, Gs, also stimulates dihydropyridine-sensitive Ca2+ channels. Evidence for direct regulation independent of phosphorylation by cAMP-dependent protein kinase or stimulation by a dihydropyridine agonist.Published by Elsevier ,1988
- Chronic ethanol causes heterologous desensitization of receptors by reducing αsmessenger RNANature, 1988
- Subcellular localization and quantitation of the major neutrophil pertussis toxin substrate, Gn.The Journal of cell biology, 1988
- Antibodies which recognize the C-terminus of the inhibitory guanine-nucleotide-binding protein (Gi) demonstrate that opioid peptides and foetal-calf serum stimulate the high-affinity GTPase activity of two separate pertussis-toxin substratesBiochemical Journal, 1988
- Identification of receptor contact site involved in receptor–G protein couplingNature, 1987
- Altered Gs and adenylate cyclase activity in human GH-secreting pituitary adenomasNature, 1987
- Identification of the lesion in the stimulatory GTP‐binding protein of the uncoupled S49 lymphomaFEBS Letters, 1987
- Chemically induced hypothyroidism produces elevated amounts of the α subunit of the inhibitory guanine nucleotide binding protein (Gi) and the β subunit common to all G-proteinsBiochemical Journal, 1987