Kupffer cell inactivation alleviates ethanol-induced steatosis and CYP2E1 induction but not inflammatory responses in rat liver
- 1 June 2000
- journal article
- Published by Elsevier in Journal of Hepatology
- Vol. 32 (6) , 900-910
- https://doi.org/10.1016/s0168-8278(00)80094-x
Abstract
No abstract availableKeywords
This publication has 50 references indexed in Scilit:
- Effect of Chronic Coadministration of Endotoxin and Ethanol on Rat Liver Pathology and Proinflammatory and Anti–Inflammatory CytokinesHepatology, 1999
- Cytokines and the liverJournal of Hepatology, 1997
- Alcohol‐Induced Expression of the CD14 Endotoxin Receptor Protein in Rat Kupffer CellsAlcohol, Clinical and Experimental Research, 1997
- Antibiotics prevent liver injury in rats following long-term exposure to ethanolGastroenterology, 1995
- The role of tumor necrosis factor-α in acute endotoxin-induced hepatotoxicity in ethanol-fed ratsHepatology, 1994
- Lactobacillus Feeding Reduces Endotoxemia and Severity of Experimental Alcoholic Liver (Disease)Experimental Biology and Medicine, 1994
- Role of cytochrome P4502E1 in alcoholic liver disease pathogenesisAlcohol, 1993
- Ethanol-inducible cytochrome P4502E1: Genetic polymorphism, regulation, and possible role in the etiology of alcohol-induced liver diseaseAlcohol, 1993
- Rat liver microsomal NADPH-supported oxidase activity and lipid peroxidation dependent on ethanol-inducible cytochrome P-450 (P-450IIE1)Biochemical Pharmacology, 1989
- Endotoxemia in patients with alcoholic and non-alcoholic cirrhosis and in subjects with no evidence of chronic liver disease following acute alcohol excessJournal of Hepatology, 1987