Genetic p53 Deficiency Partially Rescues the Adrenocortical Dysplasia Phenotype at the Expense of Increased Tumorigenesis
- 1 June 2009
- journal article
- research article
- Published by Elsevier in Cancer Cell
- Vol. 15 (6) , 465-476
- https://doi.org/10.1016/j.ccr.2009.04.011
Abstract
No abstract availableKeywords
This publication has 65 references indexed in Scilit:
- An Extended Transcriptional Network for Pluripotency of Embryonic Stem CellsCell, 2008
- TERT Promotes Epithelial Proliferation through Transcriptional Control of a Myc- and Wnt-Related Developmental ProgramPLoS Genetics, 2008
- Melanocortin 2 receptor is required for adrenal gland development, steroidogenesis, and neonatal gluconeogenesisProceedings of the National Academy of Sciences, 2007
- Dysfunctional telomeres activate an ATM-ATR-dependent DNA damage response to suppress tumorigenesisThe EMBO Journal, 2007
- Telomere dysfunction suppresses spontaneous tumorigenesis in vivo by initiating p53‐dependent cellular senescenceEMBO Reports, 2007
- A critical role for TPP1 and TIN2 interaction in high-order telomeric complex assemblyProceedings of the National Academy of Sciences, 2006
- Telomeres and telomerase in cancer stem cellsEuropean Journal Of Cancer, 2006
- Conditional telomerase induction causes proliferation of hair follicle stem cellsNature, 2005
- Tyrosinase Gene Expression is Regulated by p53Journal of Investigative Dermatology, 2002
- Tumor spectrum analysis in p53-mutant miceCurrent Biology, 1994