Myocardial ischaemia: What happens to the coronary arteries?

This publication has 39 references indexed in Scilit:

• Allopurinol and amlodipine improve coronary vasodilatation after myocardial ischaemia and reperfusion in anaesthetized dogs
  British Journal of Pharmacology, 1993
• Diminished basal nitric oxide release after myocardial ischemia and reperfusion promotes neutrophil adherence to coronary endothelium.
  Circulation Research, 1993
• Endothelium-derived relaxing factor modulates platelet aggregation in an in vivo model of recurrent platelet activation.
  Circulation Research, 1992
• Acute impairment of endothelium-dependent relaxations to aggregating platelets following reperfusion injury in canine coronary arteries.
  Circulation Research, 1990
• Alteration of endothelium-dependent distribution of myocardial blood flow after coronary occlusion and reperfusion.
  Circulation, 1990
• Impaired vasodilatation of epicardial coronary arteries and resistance vessels following myocardial ischemia and reperfusion in anesthetized dogs
  Coronary Artery Disease, 1990
• Protection by superoxide dismutase from myocardial dysfunction and attenuation of vasodilator reserve after coronary occlusion and reperfusion in dog.
  Circulation Research, 1989
• Impaired canine coronary vasodilator response to acetylcholine and bradykinin after occlusion-reperfusion.
  Circulation Research, 1989
• Local platelet activation causes vasoconstriction of large epicardial canine coronary arteries in vivo. Thromboxane A2 and serotonin are possible mediators.
  Circulation, 1989
• Reduction in coronary vasodilator reserve following coronary occlusion and reperfusion in anesthetized dog: Role of endothelium-derived relaxing factor, myocardial neutrophil infiltration and prostaglandins
  Journal of Molecular and Cellular Cardiology, 1988