EFFECT OF SANGUINARINE ON VENTRICULAR REFRACTORINESS
- 1 January 1980
- journal article
- letter
- Vol. 29 (2) , 377-380
Abstract
The effect of the Na+K+ATPase inhibitor sanguinarine, derived from the root of Sanguinaria canadadensis, on the refractory period of the porcine ventricle was studied. Measurements of ventricular refractoriness and strength-interval curves were obtained before and after 0.5-4.0 mg/kg sanguinarine. The ventricular refractory period was prolonged at all doses studied from a control of 276 .+-. 12 ms to 318 .+-. 24 ms at 4.0 mg/kg (P < 0.01). The strength-interval curve was shifted to increased values for ventricular refractoriness from 0.5-16.0 mA. No depression of arterial pressure or left ventricular dp/dt [rate of pressure change] was observed. This study shows that sanguinarine prolongs ventricular refractoriness. This property may be useful in the treatment of ventricular arrhythmias.This publication has 3 references indexed in Scilit:
- Changes in myocardial refractory periods following ischemia in the porcine heartJournal of Electrocardiology, 1978
- Structural specificity of the NaK-ATPase inhibition by sanguinarine, an isoquinoline benzophenanthridine alkaloidBiochemical Pharmacology, 1978
- BINDING AND EFFECT OF TRITIATED QUINIDINE ON CARDIAC SUBCELLULAR ENZYME-SYSTEMS - SARCOPLASMIC-RETICULUM VESICLES, MITOCHONDRIA AND NA+, K+-ADENOSINE TRIPHOSPHATASE1977