Receptor site specificity for the acute effects of β-N-methylamino-alanine in mice
- 1 October 1990
- journal article
- research article
- Published by Elsevier in European Journal of Pharmacology
- Vol. 187 (1) , 131-134
- https://doi.org/10.1016/0014-2999(90)90350-f
Abstract
No abstract availableThis publication has 8 references indexed in Scilit:
- Selective degeneration of cerebellar cortical neurons caused by cycad neurotoxin, L‐β‐methylaminoalanine (L‐BMAA), in ratsNeuropathology and Applied Neurobiology, 1990
- Neurotoxicity ofβ-N-methylamino-l-alanine (BMAA) andβ-N-oxalylamino-l-alamine (BOAA) on cultured cortical neuronsBrain Research, 1989
- Electrophysiological studies with a 2,3-benzodiazepine muscle relaxant: GYKI 52466European Journal of Pharmacology, 1989
- Guam Amyotrophic Lateral Sclerosis-Parkinsonism-Dementia Linked to a Plant Excitant NeurotoxinScience, 1987
- Evidence that antagonism at non-NMDA receptors results in anticonvulsant actionEuropean Journal of Pharmacology, 1987
- Specific antagonism of behavioral action of “uncommon” amino acids linked to motor‐system diseasesSynapse, 1987
- The preparation of l- and d-α-amino-β-methylaminopropionic acids and the identification of the compound isolated from Cycas circinalis as the l-isomerPhytochemistry, 1968
- Epidemiologic Investigations of Amyotrophic Lateral SclerosisNeurology, 1954