A potential role for PRDM12 in the pathogenesis of chronic myeloid leukaemia with derivative chromosome 9 deletion

This publication has 9 references indexed in Scilit:

• A novel EVI1 gene family, MEL1, lacking a PR domain (MEL1S) is expressed mainly in t(1;3)(p36;q21)-positive AML and blocks G-CSF–induced myeloid differentiation
  Blood, 2003
• Quantitative PCR identifies a minimal deleted region of 120 kb extending from the Philadelphia chromosome ABL translocation breakpoint in chronic myeloid leukemia with poor outcome
  Leukemia, 2003
• Identification of a Functionally Impaired Positive Regulatory Domain I Binding Factor 1 Transcription Repressor in Myeloma Cell Lines
  The Journal of Immunology, 2003
• Altered expression of retinoblastoma protein‐interacting zinc finger gene, RIZ, in human leukaemia
  British Journal of Haematology, 2002
• Quantitative comparison of the expression of EVI1 and its presumptive antagonist, MDS1/EVI1, in patients with myeloid leukemia
  Genes, Chromosomes and Cancer, 2002
• Deletions of the derivative chromosome 9 occur at the time of the Philadelphia translocation and provide a powerful and independent prognostic indicator in chronic myeloid leukemia
  Blood, 2001
• Tumor formation and inactivation of RIZ1, an Rb-binding member of a nuclear protein–methyltransferase superfamily
  Genes & Development, 2001
• Loss of FBP function arrests cellular proliferation and extinguishes c-myc expression
  The EMBO Journal, 2000
• PFM1 (PRDM4), a New Member of the PR-Domain Family, Maps to a Tumor Suppressor Locus on Human Chromosome 12q23–q24.1
  Genomics, 1999