A Signal for the Caspase-1 Inflammasome Free of TLR
- 1 April 2007
- Vol. 26 (4) , 383-385
- https://doi.org/10.1016/j.immuni.2007.04.005
Abstract
No abstract availableKeywords
This publication has 10 references indexed in Scilit:
- Interleukin-1–Receptor Antagonist in Type 2 Diabetes MellitusNew England Journal of Medicine, 2007
- Pannexin-1-Mediated Recognition of Bacterial Molecules Activates the Cryopyrin Inflammasome Independent of Toll-like Receptor SignalingImmunity, 2007
- Caspase-1-dependent processing of pro-interleukin-1β is cytosolic and precedes cell deathJournal of Cell Science, 2007
- Pannexin-1 Couples to Maitotoxin- and Nigericin-induced Interleukin-1β Release through a Dye Uptake-independent PathwayJournal of Biological Chemistry, 2007
- Inflammatory caspases and inflammasomes: master switches of inflammationCell Death & Differentiation, 2006
- Neonatal-Onset Multisystem Inflammatory Disease Responsive to Interleukin-1β InhibitionNew England Journal of Medicine, 2006
- Nalp1b controls mouse macrophage susceptibility to anthrax lethal toxinNature Genetics, 2006
- Role of interleukin-1 (IL-1) in the pathogenesis of systemic onset juvenile idiopathic arthritis and clinical response to IL-1 blockadeThe Journal of Experimental Medicine, 2005
- Phospholipases C and A2control lysosome-mediated IL-1β secretion: Implications for inflammatory processesProceedings of the National Academy of Sciences, 2004
- Biologic basis for interleukin-1 in diseaseBlood, 1996